Current diagnostic methods lack the spatial resolution and molecular specificity to precisely map fibrotic progression within complex tissue architectures. Improving the accuracy of these assessments enables earlier intervention and more reliable monitoring of disease pathology.

Genetic mutations and aberrant pre-messenger RNA splicing lead to non-functional protein production. Correcting these errors prevents disease progression and restores cellular homeostasis.

Inconsistent identification of specific pathological states across diverse datasets. Resolving this improves diagnostic precision and clinical decision-making.